Understanding the aging process is crucial as global populations, especially in developed countries, get older. This knowledge can help alleviate healthcare burdens and promote strategies for healthier aging. Research indicates that preventing excessive senescence, which contributes to various pathologies, can enhance tissue function and homeostasis.
We have a robust background in studying how senescent cells remain viable despite underlying damage. Our current goal is to connect these findings to defective tissue damage responses, particularly during aging and fibrosis. Our research focuses on:

1.- Examining the connections between cellular senescence and fibrosis through the extracellular matrix.
2.- Investigating senescence dynamics in vivo following organ injury.
3.- Developing therapies targeting senescent cells to improve age-related conditions.

These efforts aim to deepen our understanding of the roles of cellular senescence in multiple contexts, ultimately seeking to mitigate age-dependent functional declines.